Intramedullary Spinal Cord Tumors by G. Fischer

By G. Fischer

College of Lyons, France. textual content at the advances of intramedullary spinal twine tumor remedy, for neurosurgeons. Discusses diagnostic and healing difficulties, elements of tumors in accordance with histological forms, and the result of 171 spinal twine tumor surgical procedures. 30 individuals, no U.S. DNLM: Spinal wire Neoplasms.

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Intramedullary Spinal Cord Tumors

College of Lyons, France. textual content at the advances of intramedullary spinal twine tumor therapy, for neurosurgeons. Discusses diagnostic and healing difficulties, features of tumors based on histological forms, and the result of 171 spinal twine tumor surgical procedures. 30 individuals, no U. S. DNLM: Spinal wire Neoplasms.

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Extra resources for Intramedullary Spinal Cord Tumors

Sample text

We have described a group of astrocytomas as being dysplastic, since they seem to us to be more hamartomatous than tumoral in nature. Their course is similar to that of pilocytic astrocytomas. For these two reasons, they can be rated as grade I astrocytomas. Material and Methods Selection criteria and discussion of diagnoses. Our personal experience is based on an analysis by three neuropathologists (A. Jouvet, I. Salmon, M. Tommasi), using light microscopy, of specimens from 214 operations. Fifteen cases were excluded: two because the pathological process was not neoplastic (one demyelination, one ischemia); two because the final histological examination showed no evidence of tumor; and 11 because the tumors were not intra­ medullary in their location.

In one of them [ 1 621, the lesion extended to L2, and it may well have had an impact on the N22 response. either by a mass effect or because the extension down the spinal cord was lower than previously Diagnosis thought by the surgeon. This explanation was not the case with the second patient {160J, who presented with a n o cavernoma; in this patient, a falsely abnor­ mal N22 potential seems the most likely explanation. Abnormalities ofintraspinai conduction (prolong­ ed N22 - P39 interval or absent P39).

Upper limb SEPs obtained by stimulation of the median or ulnar nerve at the wrist ( Fig. 15). a) Peak latencies of the N9 and P9 potentials obtained from the supraclavicular fossa and the scalp ( activity of the brachial plexus trunks ); b) peak latencies of the P14 ( afferent volleys reaching the cervicomedullary junc­ tion) and N20 (first response of the primary sensory cortex) potentials obtained from the scalp; c) P9- P14 (conduction time in the posterior columns of the cervical region) and P14-N20 ( conduction time in the lemniscal tract and thalamocortical tract) intervals; d) P9 - P14 amplitude ratio (dispersion index of afferent volleys in the posterior columns of the cervical region); e) amplitude and latency of the N13 cervical potential ( response of the dorsal horn of the cervical spinal cord).

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